Lymphoma and Electrical Pollution

Dr. Weeks’ Comment: Read the following new study from Israel bearing in mind that radar utilizes frequencies in the same range as cell phones and Wi-Fi so anything damning about radar is equally damning about cell phones and wifi.

For more lifesaving information about protecting yourself and your loved ones (especially children) from this 21st century plague,  search at  the term “electric” or “electrical pollution”.  Note that many “elite” endorse eugenics and population control  (they think there are too many of the other people on earth…time for a winnowing of the population) so tell me, what is the most elegant and lethal smart bomb which kills people while leaving the infrastructure intact? Correct. Your sexy and addictive cell phone. Remember, you violated the terms of engagement with the manufacturer of your cell phone when you turned it on and held it. You agreed never to have the cell phone turned on and closer than 5/8th of an inch from your body.  So by simply holding the cellphone (let along holding it up to your ear/skull/brian), you violate the terms of engagement and can no longer hold the manufacturer liable when you or your child gets brain cancer.   Or as this study shows, lymphoma.


Lymphoma is increasing at a rate of 3% per year in the US
Non-Hodgkins lymphoma considered an “emerging epidemic” by the National Cancer Institute
Lymphoma rates increased in those with psoriasis
(psoriasis is a type of skin rash.  Have you noticed the increased commercials lately for pharmaceutical products for eczema and psoriasis?)
Multiple Myeloma increasing world-wide, especially in the US.  From 1990 to 2016, incident cases of myeloma increased by 126% globally and deaths increased 94%.
Leukemia is up 35% from 1975 to 2016. likely due to an environmental cause, and it is the most common childhood cancer
A recently published 2018 Israeli study on incidence of cancer in the military  in 3 countries from exposure to radar and communication equipment found a consistent association of radiofrequency radiation (RFR) and highly elevated hemolymphatic (HL) cancers such as multiple myeloma, leukemia, lymphoma, and plasma cell tumors  “suggests a cause-effect relationship between RFR and HL cancers in military/occupational settings”  (Radar utilizes frequencies in the same range as cell phones and Wi-Fi.)
Higher than expected frequencies of hematolymphatic cancers were found in military populations exposed to high levels of RF radiation from radar compared to unexposed populations; results were statistically significant.

Full Text of study

Methods: We extended an analysis of an already-reported case series of patients with cancer previously exposed to whole-body prolonged RFR, mainly from communication equipment and radar. We focused on hematolymphatic (HL) cancers. We used analysis by percentage frequency (PF) of a cancer type, which is the proportion of a specific cancer type relative to the total number of cancer cases. We also examined and analyzed the published data on three other cohort studies from similar military settings from different countries.
Results: The PF of HL cancers in the case series was very high, at 40% with only 23% expected for the series age and gender profile, confidence interval CI95%: 26–56%, p < 0.01, 19 out of 47 patients had HL cancers. We also found high PF for multiple primaries. As for the three other cohort studies: In the Polish military sector, the PF of HL cancers was 36% in the exposed population as compared to 12% in the unexposed population, p < 0.001. In a small group of employees exposed to RFRin Israeli defense industry, the PF of HL cancers was 60% versus 17% expected for the group age and gender profile, p < 0.05. In Belgian radar battalions the HL PF was 8.3% versus 1.4% in the control battalions as shown in a causes of deaths study and HL cancer mortality rate ratio was 7.2 and statistically significant. Similar findings were reported on radio amateurs and Korean war technicians. Elevated risk ratios were previously reported in most of the above studies.
 Conclusions: The consistent association of RFR and highly elevated HL cancer risk in the four groups spread over three countries, operating different RFR equipment types and analyzed by different research protocols, suggests a cause-effect relationship between RFR and HL cancers in military/occupational settings. While complete measurements of RFR exposures were not available and rough exposure assessments from patients interviews and from partial exposure data were used instead, we have demonstrated increased HL cancers in occupational groups with relatively high RFR exposures. Our findings, combined with other studies, indicate that exposures incurred in the military settings evaluated here significantly increased the risk of HL cancers. Accordingly, the RFR military exposures in these occupations should be substantially reduced and further efforts should be undertaken to monitor and measure those exposures and to follow cohorts exposed to RFR for cancers and other health effects. Overall, the epidemiological studies on excess risk for HL and other cancers together with brain tumors in cellphone users and experimental studies on RFR and carcinogenicity make a coherent case for a cause-effect relationship and classifying RFR exposure as a human carcinogen (IARC group 1).

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